@Jay Hemdal has on numerous occasions discussed the impact of propagule pressure on the frequency and severity of ich and other parasite infections in reef tanks.
Very few react to these comments, perhaps because they don't agree, or more likely because they haven't taken the time to try to understand it.
I think an additional, related hypothesis should be advanced.
The noticeable absence of ich on ocean fish is due to the absence of propagule pressure, not immunity acquired from exposure to the parasite.
Think about it.
The ich parasite requires a host to complete its life cycle. Yet, when the prototomont drops from the fish, as it drops to the bottom it is carried by current to who knows where. At the same time, the host fish swims away to who knows where. Just imagine what the concentration of tomonts and theronts would need to be to successfully attach to fish in millions of gallons of water.
1 cubic mile of water = 147,197,952,000 cubic feet =
1,101,117,147,352 gallons.
OK, so perhaps the average reef fish remains in an area the size of a football field to a depth of 25 feet.
That's 1,440,000 cubic feet; only 10,771,200 gallons in a football field 25 feet deep.
So, one Trophont (spot on fish) creates one Protomont which creates one Tomont which creates 200 tomonts which creates 200 Theronts which must attach to a fish host within 48 hours or starve to death.
Where is that life cycle likely to be successful, in the ocean or in an aquarium?
My unproven theory: propagule pressure is responsible for the frequency and severity of parasite infection of aquarium fish; the lack of propagule pressure, not immunity, is responsible for the seeming absence of parasite infection in the ocean.
I would also suggest that most parasitic infections that make it to aquariums most likely occur because of exposure to parasites during the fish's journey through the supply chain rather that infection while in the ocean.